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عمومی::
وازودیلاتورهای مغزی (متسعکنندههای عروق مغزی)
Glutamate, released with increased neuronal activity, results in the synthesis and release of NO, a potent cerebral vasodilator that plays an important role in neurovascular coupling.
The mechanisms medi- ating cerebral vasodilation during hypoxia may include neurogenic effects initiated by peripheral and neuraxial
Both hemodilution and hypoxemic hypoxia lead to cerebral vasodilation and an increase in CBF.
is induced with a cerebral vasodilator, CBF is maintained at lower MAP values than when induced by either hem- orrhage or a noncerebral vasodilator.
Nimodipine in human subjects does, however, blunt auto- regulation moderately.48 Intraarterial nimodipine for the treatment of cerebral vasospasm after subarachnoid hem- orrhage increased regional CBF significantly provided MAP was maintained, indicating that nimodipine is a cerebral vasodilator.49
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